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The corticosteroids that help treat rheumatoid arthritis (RA) are not the same as the steroids an athlete might take to build muscle; these steroids are meant to boost both the number of cells that are able to repair themselves when pain is caused, as well as the number of cells that can produce enough immune proteins to fight off an infection – or even cancer. So it is possible that the number of T cells that the athlete produces to combat arthritis and other ailments might be insufficient or even negative. Researchers say the new study supports earlier, more rigorous studies by others, which showed an important correlation between the production of T cells and the number of people diagnosed with the disease. "We thought these mechanisms would be undergirding RA, and certainly it seems to be a consequence of the underlying pathology," said Dr. Robert J. Lee, professor of medicine at the University of Maryland School of Medicine and co-author on the new study. "It makes some things much worse and some things much better and it has not been well understood yet." (Dr. Lee is also a member of the editorial board of BMJ, the British medical journal.) Accordingly, the new authors argue that, at best, the new study "is an initial validation of previous findings that could lead to improved diagnosis, more effective treatment and a healthier quality of life." Other experts agree, but for good reason. First and foremost it adds to the mounting evidence, recently published in the journal Nature Genetics, that the number of T cell-producing cells in the body, known as Tregs, play an important role in the autoimmune disease. Another recent study of rheumatoid arthritis researchers led by lead author Mark J. Giannides, director of Duke University in Durham, N.C., showed that Tregs can help to protect against damage from infections. In the current study, Tregs in humans were measured in the blood; this allowed them to isolate the populations that produce more T cells. This, Lee said, was important because the ability of a given Treg population to produce more T cells is largely determined by the genetic makeup of that population. Lee and his colleagues also looked at the T cells' ability to produce inflammation-fighting protein called interferon gamma. The researchers identified a gene – called IL-36 – which they believe helps to boost IL-36 production. The gene also increased the body's production of interferon beta, which inhibits the formation of white blood cells by Treg cells. Related Article: